Michal Kidacki, MD/PhD
Clinical Fellow
Research & Publications
Biography
Research Summary
My love for research was ignited in the lab of Dr. Vonderhaar in the Mammary Biology and Tumorigenesis Lab at NIH. We studied the interactions between immune cells and stroma of the breast tissue. I completed my PhD at Penn State University in the department of Pathology studying p120-catenin and it's effects on tumorigenesis in squamous cancers of the esophagus and oral cavity. During my last two years of medical school I worked with Dr. Amanda Nelson in the department of Dermatology studying Hidradenitis Suppurativa and its' Invasive Proliferative Gelatinous Mass.
Coauthors
Research Interests
Immune System; Hair Follicle
Selected Publications
- Immune Inhibitory Molecule PD-1 Homolog (VISTA) Colocalizes with CD11b Myeloid Cells in Melanoma and Is Associated with Poor OutcomesVesely M, Kidacki M, Gaule P, Gupta S, Chan N, Han X, Yeung J, Chen L. Immune Inhibitory Molecule PD-1 Homolog (VISTA) Colocalizes with CD11b Myeloid Cells in Melanoma and Is Associated with Poor Outcomes. Journal Of Investigative Dermatology 2023, 144: 106-115.e4. PMID: 37562584, DOI: 10.1016/j.jid.2023.07.008.
- Iatrogenic calcinosis cutis from extravasated phosphate-containing solution treated with topical sodium thiosulfateXie CB, Kidacki M, Ring N, Panse G, Leventhal JS. Iatrogenic calcinosis cutis from extravasated phosphate-containing solution treated with topical sodium thiosulfate. JAAD Case Reports 2021, 17: 31-33. PMID: 34692964, PMCID: PMC8517711, DOI: 10.1016/j.jdcr.2021.09.013.
- Twist2 is NFkB-responsive when p120-catenin is inactivated and EGFR is overexpressed in esophageal keratinocytesLehman H, Kidacki M, Stairs D. Twist2 is NFkB-responsive when p120-catenin is inactivated and EGFR is overexpressed in esophageal keratinocytes. Scientific Reports 2020, 10: 18829. PMID: 33139779, PMCID: PMC7608670, DOI: 10.1038/s41598-020-75866-0.
- E‐cadherin and p120ctn protein expression are lost in hidradenitis suppurativa lesionsNelson AM, Cong Z, Gettle SL, Longenecker AL, Kidacki M, Kirby JS, Adams DR, Stairs DB, Danby FW. E‐cadherin and p120ctn protein expression are lost in hidradenitis suppurativa lesions. Experimental Dermatology 2019, 28: 867-871. PMID: 31107992, DOI: 10.1111/exd.13973.
- 1028 Deciphering the mechanism of action of isotretinoin in acne patients using next gene sequencing technologiesThiboutot D, Kidacki M, Sarfo A, Schneider A, Albert I, Longenecker A, Cong Z, Nelson A. 1028 Deciphering the mechanism of action of isotretinoin in acne patients using next gene sequencing technologies. Journal Of Investigative Dermatology 2019, 139: s178. DOI: 10.1016/j.jid.2019.03.1104.
- ‘Invasive proliferative gelatinous mass’ of hidradenitis suppurativa contains distinct inflammatory componentsKidacki M, Cong Z, Flamm A, Helm K, Danby FW, Nelson AM. ‘Invasive proliferative gelatinous mass’ of hidradenitis suppurativa contains distinct inflammatory components. British Journal Of Dermatology 2019, 181: 192-193. PMID: 30597519, DOI: 10.1111/bjd.17541.
- An Immunohistopathologic Study to Profile the Folate Receptor Beta Macrophage and Vascular Immune Microenvironment in Giant Cell Arteritis.Albano-Aluquin S, Malysz J, Kidacki M, Ratnam M, Olsen NJ. An Immunohistopathologic Study to Profile the Folate Receptor Beta Macrophage and Vascular Immune Microenvironment in Giant Cell Arteritis. Journal Of Visualized Experiments 2019 PMID: 30799840, DOI: 10.3791/58713.
- An Immunohistopathologic Study to Profile the Folate Receptor Beta Macrophage and Vascular Immune Microenvironment in Giant Cell ArteritisAlbano-Aluquin S, Malysz J, Kidacki M, Ratnam M, Olsen N. An Immunohistopathologic Study to Profile the Folate Receptor Beta Macrophage and Vascular Immune Microenvironment in Giant Cell Arteritis. Journal Of Visualized Experiments 2019 DOI: 10.3791/58713-v.
- Pseudohyperphosphatemia in a patient with incidentally identified progression of smoldering myelomaFrancis ER, Chen F, Kidacki M, Miller R, Alkhasoneh M, Talamo G, Straub M, Klinefelter K, Kodali N, Zhu Y. Pseudohyperphosphatemia in a patient with incidentally identified progression of smoldering myeloma. Clinica Chimica Acta 2018, 487: 306-308. PMID: 30315756, DOI: 10.1016/j.cca.2018.10.016.
- NFkB hyperactivation causes invasion of esophageal squamous cell carcinoma with EGFR overexpression and p120-catenin down-regulationLehman HL, Kidacki M, Warrick JI, Stairs DB. NFkB hyperactivation causes invasion of esophageal squamous cell carcinoma with EGFR overexpression and p120-catenin down-regulation. Oncotarget 2018, 9: 11180-11196. PMID: 29541406, PMCID: PMC5834278, DOI: 10.18632/oncotarget.24358.
- p120-Catenin Downregulation and PIK3CA Mutations Cooperate to Induce Invasion through MMP1 in HNSCCKidacki M, Lehman HL, Green MV, Warrick JI, Stairs DB. p120-Catenin Downregulation and PIK3CA Mutations Cooperate to Induce Invasion through MMP1 in HNSCC. Molecular Cancer Research 2017, 15: 1398-1409. PMID: 28637905, DOI: 10.1158/1541-7786.mcr-17-0108.
- Tu1175 NFkB Regulates the Invasive Potential of Esophageal Squamous Cell Carcinoma Downstream of EGFR and p120ctnLehman H, Welsh P, Kidacki M, Warrick J, Stairs D. Tu1175 NFkB Regulates the Invasive Potential of Esophageal Squamous Cell Carcinoma Downstream of EGFR and p120ctn. Gastroenterology 2016, 150: s854-s855. DOI: 10.1016/s0016-5085(16)32885-2.
- Higher iron in the red nucleus marks Parkinson's dyskinesiaLewis MM, Du G, Kidacki M, Patel N, Shaffer ML, Mailman RB, Huang X. Higher iron in the red nucleus marks Parkinson's dyskinesia. Neurobiology Of Aging 2012, 34: 1497-1503. PMID: 23177595, PMCID: PMC3570638, DOI: 10.1016/j.neurobiolaging.2012.10.025.
- Paracrine interactions between primary human macrophages and human fibroblasts enhance murine mammary gland humanization in vivoFleming JM, Miller TC, Kidacki M, Ginsburg E, Stuelten CH, Stewart DA, Troester MA, Vonderhaar BK. Paracrine interactions between primary human macrophages and human fibroblasts enhance murine mammary gland humanization in vivo. Breast Cancer Research 2012, 14: r97. PMID: 22731827, PMCID: PMC3446360, DOI: 10.1186/bcr3215.