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How Depression Affects the Brain

December 16, 2025

Transcript

  • 00:08The current standard of care
  • 00:10for the treatment of depression
  • 00:13is based on what we
  • 00:14call the monoamine
  • 00:15deficiency hypothesis.
  • 00:17Essentially presuming
  • 00:19that one of three neurotransmitters
  • 00:21in the brain is deficient
  • 00:23or under active. Neurotransmitters can
  • 00:25be thought of as the
  • 00:26chemical messengers within the brain.
  • 00:28It's what helps one cell
  • 00:29in the brain communicate with
  • 00:30another to pass that message
  • 00:32along. But the reality is
  • 00:33there are more than a
  • 00:34hundred neurotransmitters
  • 00:35in the brain and billions
  • 00:37of connections between neurons.
  • 00:39So we know that that's
  • 00:41a limited hypothesis.
  • 00:48For decades,
  • 00:49we thought that the primary
  • 00:52pathology, the primary cause of
  • 00:53depression was some abnormality in
  • 00:55these neurotransmitters,
  • 00:56specifically serotonin or norepinephrine.
  • 00:59However, norepinephrine
  • 01:01and serotonin
  • 01:02did not seem to be
  • 01:03able to account for the
  • 01:05symptoms of depression in people
  • 01:07who had major depression.
  • 01:09Instead,
  • 01:10the chemical messengers
  • 01:12between the nerve cells in
  • 01:14the higher centers of the
  • 01:15brain involved in
  • 01:17regulating mood and emotion, which
  • 01:20include glutamate and GABA,
  • 01:22were
  • 01:23possibilities
  • 01:24as alternative causes
  • 01:26for the symptoms of depression.
  • 01:28We know that these two,
  • 01:30which are the most ubiquitous
  • 01:31and abundant
  • 01:33neurotransmitters in the brain,
  • 01:35actually regulate
  • 01:36how the brain is
  • 01:38changing over time and adapting.
  • 01:40When you are exposed to
  • 01:42severe and chronic stress, like
  • 01:43people experience when they have
  • 01:45depression,
  • 01:46you lose some of the
  • 01:48connections between the nerve cells.
  • 01:50And the communication in these
  • 01:52circuits
  • 01:53becomes inefficient
  • 01:55and noisy.
  • 01:57Because of the
  • 01:58noisy communication
  • 02:00in the circuits involved
  • 02:02in regulating mood and emotion.
  • 02:05We think that the loss
  • 02:07of these synaptic connections
  • 02:11contributes to the biology of
  • 02:12depression.
  • 02:19It's critical to understand the
  • 02:21neurobiology
  • 02:22of depression and how the
  • 02:24brain plays a role in
  • 02:25that for two main reasons.
  • 02:27One, it helps us understand
  • 02:29how the disease
  • 02:30develops and progresses
  • 02:32and we can start to
  • 02:33target treatments based on that.
  • 02:35There are clear differences between
  • 02:37a healthy brain and a
  • 02:38depressed brain. And the exciting
  • 02:40thing is when you treat
  • 02:41that depression
  • 02:44effectively,
  • 02:45the brain goes back to
  • 02:46looking like a healthy brain.
  • 02:47We recognize that the treatment
  • 02:49for depression is a long
  • 02:50term process because for many
  • 02:52people depression is a long
  • 02:54term disorder.
  • 02:56So we need new treatments.
  • 02:59We've needed new ways to
  • 03:01approach depression
  • 03:02for people that haven't responded
  • 03:04well to their prior treatments.
  • 03:06We are
  • 03:07in a new era of
  • 03:09psychiatry. Psychiatry. This is a
  • 03:11paradigm shift
  • 03:12away from
  • 03:15a model of monoamaminergic
  • 03:18deficiency
  • 03:19to
  • 03:20a fuller understanding
  • 03:22of the brain as a
  • 03:23complex
  • 03:25neurochemical
  • 03:29organ.