Madison Nashu, MD, MS
Instructor of Emergency MedicineDownloadHi-Res Photo
Cards
Contact Info
About
Titles
Instructor of Emergency Medicine
Departments & Organizations
Education & Training
- MD
- University of Toledo College of Medicine and Life Sciences (2020)
- MS
- University of Cincinnati, Molecular, Cellular, and Biochemical Pharmacology (2015)
- BSc
- The Ohio State University, Biology (2012)
- BS
- The Ohio State University, Psychology (2012)
Research
Research at a Glance
Yale Co-Authors
Frequent collaborators of Madison Nashu's published research.
Publications Timeline
A big-picture view of Madison Nashu's research output by year.
Alina Tsyrulnik, MD
Katja Goldflam, MD
6Publications
121Citations
Publications
2025
263 Duration and Frequency of Methamphetamine Use and Associated Reduced Cardiac Ejection Fraction
Nashu M, Khan Z, Lam J, Saadat S, Goubert R, Almog R, Hsu E, Guy M, Fox J. 263 Duration and Frequency of Methamphetamine Use and Associated Reduced Cardiac Ejection Fraction. Annals Of Emergency Medicine 2025, 86: s114. DOI: 10.1016/j.annemergmed.2025.06.278.Peer-Reviewed Original ResearchQuick to connect: a comparison of virtual and in-person speed mentoring for emergency medicine medical students, residents by faculty at a national conference
Sun W, Goldflam K, Pennington Z, Derks L, Coates W, Nashu M, Hossin T, Clark A, Tsyrulnik A, Linden J. Quick to connect: a comparison of virtual and in-person speed mentoring for emergency medicine medical students, residents by faculty at a national conference. International Journal Of Emergency Medicine 2025, 18: 42. PMID: 40033204, PMCID: PMC11877889, DOI: 10.1186/s12245-025-00860-0.Peer-Reviewed Original ResearchConceptsSociety for Academic Emergency MedicineSpeed mentoringIn-personFree-text questionsAcademic Emergency MedicineResultsThe response rateIn-person mentoringModel of mentorshipExact testEmergency medicineThematic analysisMedical studentsViewpoints of participantsFischer's exact testMethodsMedical studentsParticipantsFaculty mentorsCOVID-19 pandemicProfessional growthResponse rateVirtual format
2018
Tumor Cell Autonomous RON Receptor Expression Promotes Prostate Cancer Growth Under Conditions of Androgen Deprivation
Brown NE, Paluch AM, Nashu MA, Komurov K, Waltz SE. Tumor Cell Autonomous RON Receptor Expression Promotes Prostate Cancer Growth Under Conditions of Androgen Deprivation. Neoplasia 2018, 20: 917-929. PMID: 30121008, PMCID: PMC6098205, DOI: 10.1016/j.neo.2018.07.003.Peer-Reviewed Original ResearchCitationsAltmetricMeSH Keywords and ConceptsMeSH KeywordsAndrogensAnimalsApoptosisbeta CateninBiomarkersCell ProliferationGene Expression Regulation, NeoplasticHumansImmunohistochemistryMaleMiceNF-kappa BProstatic NeoplasmsProstatic Neoplasms, Castration-ResistantReceptor Protein-Tyrosine KinasesReceptors, AndrogenSignal TransductionTranscriptional Regulator ERGConceptsCastration-resistant prostate cancerAndrogen receptor reactivationProstate cancerAndrogen deprivationReceptor reactivationAndrogen receptor nuclear localizationHormone-refractory prostate cancerProstate cancer cell growthAR-responsive genesCurrent treatment strategiesCastration-resistant growthProstate cancer growthRON receptor tyrosine kinaseProstate cancer progressionHuman hormone-refractory prostate cancersCancer cell growthProstate cancer cellsMurine prostate cancer cellsDeprivation therapyCRPC therapyCRPC cellsMechanisms of resistanceAndrogen receptorTreatment strategiesRON overexpression
2016
NK cell activating receptor ligand expression in lymphangioleiomyomatosis is associated with lung function decline
Osterburg AR, Nelson RL, Yaniv BZ, Foot R, Donica W, Nashu MA, Liu H, Wikenheiser-Brokamp KA, Moss J, Gupta N, McCormack FX, Borchers MT. NK cell activating receptor ligand expression in lymphangioleiomyomatosis is associated with lung function decline. JCI Insight 2016, 1: e87270. PMID: 27734028, PMCID: PMC5053147, DOI: 10.1172/jci.insight.87270.Peer-Reviewed Original ResearchCitationsAltmetricMeSH Keywords and ConceptsConceptsLung function declineNK cellsLAM pathogenesisFunction declineNeoplastic smooth muscle cellsNK cell activating receptorsNKG2D surface expressionRare lung diseaseInnate immune systemSmooth muscle cellsExpiratory volumePulmonary functionLAM patientsLung diseaseActivating receptorsLAM lesionsImmune surveillanceImmune systemBinding protein 2Ligand expressionPulmonary nodulesLymphangioleiomyomatosisMuscle cellsPatientsCyst formation
2014
Hepatocyte growth factor-like protein is required for prostate tumor growth in the TRAMP mouse model
Vasiliauskas J, Nashu MA, Pathrose P, Starnes SL, Waltz SE. Hepatocyte growth factor-like protein is required for prostate tumor growth in the TRAMP mouse model. Oncotarget 2014, 5: 5547-5558. PMID: 24980820, PMCID: PMC4170603, DOI: 10.18632/oncotarget.2139.Peer-Reviewed Original ResearchCitationsMeSH Keywords and ConceptsConceptsTRAMP mouse modelHepatocyte growth factor-like proteinProstate cancerProstate tumorsMouse modelGrowth factor-like proteinReduced tumor vascularizationSmall prostate tumorsCancer cellsProstate tumor growthHuman prostate cancerProstate cancer cellsCancer cell proliferationVariety of cancersCritical survival signalsTRAMP miceCancer cell deathProstate tumorigenesisTumor growthTumor vascularizationFactor-like proteinTumor cellsCancerRon receptorElevated levelsThe DEK oncogene promotes cellular proliferation through paracrine Wnt signaling in Ron receptor-positive breast cancers
Privette Vinnedge LM, Benight NM, Wagh PK, Pease NA, Nashu MA, Serrano-Lopez J, Adams AK, Cancelas JA, Waltz SE, Wells SI. The DEK oncogene promotes cellular proliferation through paracrine Wnt signaling in Ron receptor-positive breast cancers. Oncogene 2014, 34: 2325-2336. PMID: 24954505, PMCID: PMC4275425, DOI: 10.1038/onc.2014.173.Peer-Reviewed Original ResearchCitationsAltmetricMeSH Keywords and ConceptsMeSH KeywordsAnimalsBreast NeoplasmsCell Line, TumorCell ProliferationChromosomal Proteins, Non-HistoneDNA-Binding ProteinsFemaleGene Expression Regulation, NeoplasticHumansMiceNeoplasms, ExperimentalOncogene ProteinsPoly-ADP-Ribose Binding ProteinsReceptor Protein-Tyrosine KinasesWnt Signaling PathwayConceptsRon receptor activationΒ-catenin signalingBreast cancerReceptor activationSecretion of WntHuman mammary epithelial MCF10A cellsCanonical β-cateninWnt/β-catenin signalingΒ-catenin activityBreast cancer oncogeneΒ-catenin nuclear translocationReceptor-positive breast cancerEtiology of recurrentMetastatic breast cancerCancer stem cell markersBreast cancer patientsTumorigenic phenotypeBreast cancer progressionDownstream targetsStem cell markersCellular growthParacrine WntHuman diseasesMCF10A cellsRon receptor