Krupakar Subramaniam
Postgraduate AssociateDownloadHi-Res Photo
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Titles
Postgraduate Associate
Appointments
Laboratory Medicine
Postgraduate AssociatePrimary
Other Departments & Organizations
- All Institutions
- Foxman Lab
- Laboratory Medicine
Education & Training
- BS
- Yale University (2025)
Research
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Overview
Medical Research Interests
Asthma; Cell Biology; Epithelial Cells; Immunity, Innate; Immunity, Mucosal; Interferons; Respiratory Mucosa
ORCID
0009-0008-1750-5626
Research at a Glance
Yale Co-Authors
Frequent collaborators of Krupakar Subramaniam's published research.
Research Interests
Research topics Krupakar Subramaniam is interested in exploring.
Bao Wang
Diane Krause, MD, PhD
Ellen F Foxman, MD, PhD
Michael Mak
Epithelial Cells
Interferons
Publications
2026
Dose-dependent activation of the Hippo pathway by Type I and Type III interferons suppresses tissue repair by human bronchial epithelial cells
Subramaniam K, Lim H, Wang B, Mihaylova V, Thompson E, Krause D, Foxman E. Dose-dependent activation of the Hippo pathway by Type I and Type III interferons suppresses tissue repair by human bronchial epithelial cells. PLOS Biology 2026, 24: e3003615. PMID: 41587178, PMCID: PMC12863674, DOI: 10.1371/journal.pbio.3003615.Peer-Reviewed Original ResearchAltmetricMeSH Keywords and ConceptsConceptsActivation of LATS1Type III interferonsHuman bronchial epithelial cellsLATS1 phosphorylationInfluenza virus infectionHippo pathwayIII interferonsBronchial epithelial cell migrationCanonical activationSignaling pathwayTissue repair functionsActivation of MST1/2Bronchial epithelial cellsPhosphorylation of LATS1Receptor signalingHippo signaling pathwayTissue repairIFN receptor signalingLATS1 activationEpithelial cellsJAK activityEpithelial cell migrationHigher concentrations of interferonInterferon receptor signalingJAK/STAT signaling
2024
Biophysical and biochemical aspects of immune cell–tumor microenvironment interactions
Benmelech S, Le T, McKay M, Nam J, Subramaniam K, Tellez D, Vlasak G, Mak M. Biophysical and biochemical aspects of immune cell–tumor microenvironment interactions. APL Bioengineering 2024, 8: 021502. PMID: 38572312, PMCID: PMC10990568, DOI: 10.1063/5.0195244.Peer-Reviewed Original ResearchCitationsAltmetricConceptsTumor microenvironmentImprove CAR T cell therapyCAR-T cell therapyCD4+ T cellsCancer cellsAdaptive immune systemDistribution of cancer cellsCD8+T cellsImmunosuppressive responsePro-tumorImmune cellsMicroenvironment interactionsCell therapyExtracellular matrixCancer progressionImmune systemTumorCells-thatSpatial distribution of cancer cellsCancerCellsCD8CD4Therapy
Academic Achievements & Community Involvement
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