Lydia Grmai, PhD
Associate Research ScientistDownloadHi-Res Photo
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About
Titles
Associate Research Scientist
Appointments
Comparative Medicine
Associate Research ScientistPrimary
Other Departments & Organizations
Education & Training
- Postdoctoral Associate
- University of Pittsburgh School of Medicine (2024)
- Postdoctoral Associate
- The Johns Hopkins University (2021)
- PhD
- New York University School of Medicine, Biochemistry & Molecular Pharmacology (2017)
- BS
- University of Maryland, Baltimore County, Biochemistry & Molecular Biology (2011)
Research
Overview
Medical Research Interests
Adipose Tissue; Drosophila melanogaster; Endocrinology; Fertility; Gametogenesis; Gonadal Hormones; Reproduction; Sex Differentiation
Public Health Interests
Aging; Metabolism
ORCID
0000-0001-5465-7401- View Lab Website
Grmai Lab
Research at a Glance
Publications Timeline
A big-picture view of Lydia Grmai's research output by year.
7Publications
Publications
2024
Integrated stress response signaling acts as a metabolic sensor in fat tissues to regulate oocyte maturation and ovulation.
Grmai L, Michaca M, Lackner E, Nampoothiri V P N, Vasudevan D. Integrated stress response signaling acts as a metabolic sensor in fat tissues to regulate oocyte maturation and ovulation. Cell Rep 2024, 43: 113863. PMID: 38457339, DOI: 10.1016/j.celrep.2024.113863.Peer-Reviewed Original Research
2022
The Regulation of Germline Sex Determination in Drosophila by Sex lethal.
Grmai L, Pozmanter C, Van Doren M. The Regulation of Germline Sex Determination in Drosophila by Sex lethal. Sex Dev 2022, 16: 323-328. PMID: 35259743, DOI: 10.1159/000521235.Peer-Reviewed Educational Materials
2021
Proliferative stem cells maintain quiescence of their niche by secreting the Activin inhibitor Follistatin.
Herrera SC, Sainz de la Maza D, Grmai L, Margolis S, Plessel R, Burel M, O'Connor M, Amoyel M, Bach EA. Proliferative stem cells maintain quiescence of their niche by secreting the Activin inhibitor Follistatin. Dev Cell 2021, 56: 2284-2294.e6. PMID: 34363758, DOI: 10.1016/j.devcel.2021.07.010.Peer-Reviewed Original ResearchTranscriptomic analysis of feminizing somatic stem cells in the Drosophila testis reveals putative downstream effectors of the transcription factor Chinmo.
Grmai L, Harsh S, Lu S, Korman A, Deb IB, Bach EA. Transcriptomic analysis of feminizing somatic stem cells in the Drosophila testis reveals putative downstream effectors of the transcription factor Chinmo. G3 (Bethesda) 2021, 11 PMID: 33751104, DOI: 10.1093/g3journal/jkab067.Peer-Reviewed Original Research
2018
Chinmo prevents transformer alternative splicing to maintain male sex identity.
Grmai L, Hudry B, Miguel-Aliaga I, Bach EA. Chinmo prevents transformer alternative splicing to maintain male sex identity. PLoS Genet 2018, 14: e1007203. PMID: 29389999, DOI: 10.1371/journal.pgen.1007203.Peer-Reviewed Original Research
2013
Regulation of proliferation, cell competition, and cellular growth by the Drosophila JAK-STAT pathway.
Zoranovic T, Grmai L, Bach EA. Regulation of proliferation, cell competition, and cellular growth by the Drosophila JAK-STAT pathway. JAKSTAT 2013, 2: e25408. PMID: 24069565, DOI: 10.4161/jkst.25408.Peer-Reviewed Educational Materials
2011
Proteomic pathway analysis reveals inflammation increases myeloid-derived suppressor cell resistance to apoptosis.
Chornoguz O, Grmai L, Sinha P, Artemenko KA, Zubarev RA, Ostrand-Rosenberg S. Proteomic pathway analysis reveals inflammation increases myeloid-derived suppressor cell resistance to apoptosis. Mol Cell Proteomics 2011, 10: M110.002980. PMID: 21191032, DOI: 10.1074/mcp.M110.002980.Peer-Reviewed Original Research
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