2022
Chapter 9 Regulation of salt and water balance
Holt E, Lupsa B, Lee G, Bassyouni H, Peery H. Chapter 9 Regulation of salt and water balance. 2022, 273-302. DOI: 10.1016/b978-0-12-815844-9.00009-9.Chapters
2021
Acute Myeloid Leukemia Leading to Central Diabetes Insipidus
Wojeck B, Gossmann M, Zeidan A, Inzucchi S. Acute Myeloid Leukemia Leading to Central Diabetes Insipidus. Journal Of The Endocrine Society 2021, 5: a570-a571. PMCID: PMC8090637, DOI: 10.1210/jendso/bvab048.1163.Peer-Reviewed Original ResearchCentral diabetes insipidusAcute myeloid leukemiaLeukemic infiltrationMonosomy 7Antidiuretic hormoneWhite blood cell countEVI-1 overexpressionBlood cell countAML patientsAML casesHypothalamic secretionMyeloid leukemiaDiabetes insipidusCell countPituitary glandNormal plateletsPatientsMolecular evaluationInfiltrationOverexpressionComplicationsAnemiaInsipidusLeukemiaAbnormalities
2019
Onset of pituitary hormone deficiencies in optic nerve hypoplasia: a temporal trend analysis of 32 children at Mayo Clinic
Wadams H, Gupta N, Novotny P, Tebben P. Onset of pituitary hormone deficiencies in optic nerve hypoplasia: a temporal trend analysis of 32 children at Mayo Clinic. Journal Of Pediatric Endocrinology And Metabolism 2019, 33: 139-145. PMID: 31811804, DOI: 10.1515/jpem-2019-0269.Peer-Reviewed Original ResearchConceptsOptic nerve hypoplasiaPituitary hormone deficiencyThyroid-stimulating hormoneMagnetic resonance imagingMidline abnormalitiesAdrenocorticotropic hormoneHormone deficiencyAntidiuretic hormoneDiagnosis of optic nerve hypoplasiaPresence of optic nerve hypoplasiaRetrospective chart review of patientsThyroid-stimulating hormone deficiencyGrowth hormoneChart review of patientsDiagnosis of GHPituitary hormone functionReview of patientsRetrospective chart reviewYears of ageAge 3 yearsMonths of ageNeonatal periodMedian ageFollow-upMayo Clinic
2016
Inflammasome activation by cell volume regulation and inflammation-associated hyponatremia: A vicious cycle
Kim J, Park J, Eisenhut M, Yu J, Shin J. Inflammasome activation by cell volume regulation and inflammation-associated hyponatremia: A vicious cycle. Medical Hypotheses 2016, 93: 117-121. PMID: 27372869, DOI: 10.1016/j.mehy.2016.05.018.Peer-Reviewed Original ResearchMeSH KeywordsAnimalsCell SizeCells, CulturedCentral Nervous SystemHumansHyponatremiaInappropriate ADH SyndromeInflammasomesInflammationInterleukin-1betaInterleukin-6LysosomesMiceModels, TheoreticalNeuronsNLR Family, Pyrin Domain-Containing 3 ProteinOsmolar ConcentrationOsmosisRatsRats, WistarStress, MechanicalConceptsCNS diseaseCentral nervous systemRelease of IL-1bAntidiuretic hormoneMulti-protein complexesIL-1BCell swellingSecretion of antidiuretic hormoneIL-6Nucleotide-bindingEffect of cell swellingTransient receptor potential channelsAntidiuretic hormone secretionPrevention of permanent brain damageFunction of inflammasomesProduction of antidiuretic hormoneRelease of IL-6Activate transient receptor potential channelsPosterior pituitary glandInterleukin (IL)-1bIntracellular environmentIL-1 injectionAction of prostaglandinsCell volume regulationInflammatory mediator production
2013
Inflammation and hyponatremia: an underrecognized condition?
Park J, Shin J. Inflammation and hyponatremia: an underrecognized condition? Clinical And Experimental Pediatrics 2013, 56: 519-522. PMID: 24416046, PMCID: PMC3885786, DOI: 10.3345/kjp.2013.56.12.519.Peer-Reviewed Original ResearchAntidiuretic hormoneInflammatory diseasesBrain type natriuretic peptideSodium levelsIL-6Levels of serum IL-6Elevated levels of serum IL-6Prevent potential morbidityDevelopment of hyponatremiaAssociated with severe inflammationSerum sodium levelsPatient's sodium levelsDiagnosis of hyponatremiaSerum IL-6Pediatric inflammatory diseasesInterleukin (IL) 1BC-reactive proteinDegree of inflammationMortality of patientsAssociated with inflammatory diseasesIntravenous fluid treatmentPercentage of neutrophilsADH secretionNatriuretic peptidePotential morbidity
2011
Paraneoplastic Syndrome of Inappropriate Antidiuretic Hormone Mimicking Limbic Encephalitis
Blondin NA, Vortmeyer AO, Harel NY. Paraneoplastic Syndrome of Inappropriate Antidiuretic Hormone Mimicking Limbic Encephalitis. JAMA Neurology 2011, 68: 1591-1594. PMID: 22159058, DOI: 10.1001/archneurol.2011.876.Peer-Reviewed Original ResearchConceptsLimbic encephalitisAntidiuretic hormoneParaneoplastic syndromeEctopic secretionMemory impairmentCerebrospinal fluid analysis findingsClassic limbic encephalitisParaneoplastic limbic encephalitisInappropriate antidiuretic hormoneProgressive memory impairmentAcademic medical centerMagnetic resonance imagingPositron emission tomographyOccult malignancyChronic hyponatremiaThorough workupClinical featuresNeuroendocrine tumorsDifferential diagnosisMedical CenterImmunohistochemical analysisEncephalitisResonance imagingEmission tomographyHyponatremia
2007
Gain-of-Function Mutations in the V2 Vasopressin Receptor
Rosenthal S, Gitelman S, Vargas G, Feldman B. Gain-of-Function Mutations in the V2 Vasopressin Receptor. Hormone Research In Paediatrics 2007, 67: 121-125. DOI: 10.1159/000097567.Peer-Reviewed Original ResearchSecretion of antidiuretic hormoneV2 vasopressin receptorAntidiuretic hormoneVasopressin receptorsDisorders of water balanceAbnormal water balanceArginine vasopressin levelsGain-of-function mutationsDisordered water balanceNephrogenic syndromeLaboratory featuresVasopressin levelsAssociated with such mutationsActivating mutationsWater overloadFluid homeostasisGain-of-functionLaboratory evaluationClinical disordersSyndromeClinical diseaseNSIADReceptorsMutationsHormone
2006
Nephrogenic syndrome of inappropriate antidiuresis (NSIAD): a paradigm for activating mutations causing endocrine dysfunction.
Rosenthal S, Feldman B, Vargas G, Gitelman S. Nephrogenic syndrome of inappropriate antidiuresis (NSIAD): a paradigm for activating mutations causing endocrine dysfunction. Pediatric Endocrinology Reviews : PER 2006, 4 Suppl 1: 66-70. PMID: 17261972.Peer-Reviewed Original ResearchConceptsActivating mutationsLevels of arginine vasopressinMutations of G protein-coupled receptorsAntidiuretic hormone secretionNephrogenic diabetes insipidusV2 vasopressin receptorNephrogenic syndromeV2R mutationsDiabetes insipidusArginine vasopressinClinical presentationAntidiuretic hormoneVasopressin signalingG protein-coupled receptorsEndocrine dysfunctionVasopressin receptorsHormone secretionInactivating mutationsGain-of-functionUndetectable levelsNSIADVasopressinAntidiuresisMutationsPatients
2003
Making Two Wrongs Right – Case of Using a Patient’s Diabetes insipidus to Treat HisHeart Failure: Experience with Vasopress in Antagonists from Animal Studies and Clinical Trials
Fischi M, Trichon B, Velazquez E, Blazing M, O’Conner C. Making Two Wrongs Right – Case of Using a Patient’s Diabetes insipidus to Treat HisHeart Failure: Experience with Vasopress in Antagonists from Animal Studies and Clinical Trials. Cardiology 2003, 3: 107-112. DOI: 10.1159/000071029.Peer-Reviewed Original ResearchHeart failureCongestive heart failure resultsSmooth muscle cell contractionHeart failure resultsCentral diabetes insipidusHeart failure casesMuscle cell contractionNonosmotic releaseNeurohormonal activationReceptor blockersNonischemic cardiomyopathyHypothalamic osmoreceptorsClinical trialsReceptor subtypesDiabetes insipidusAntidiuretic hormoneAnimal studiesMedical treatmentPosterior pituitaryExpression of aquaporinsPlasma osmolalityCell contractionPatientsBody waterCirculatory system
1985
Water intoxication and hyponatremic encephalopathy from the use of an oxytocin nasal spray. A case report.
Seifer DB, Sandberg EC, Ueland K, Sladen RN. Water intoxication and hyponatremic encephalopathy from the use of an oxytocin nasal spray. A case report. The Journal Of Reproductive Medicine 1985, 30: 225-8. PMID: 3923190.Peer-Reviewed Original ResearchConceptsOxytocin nasal sprayNasal sprayWater intoxicationGuillain-Barré typeSevere water intoxicationGuillain-Barré syndromeHyponatremic encephalopathyPeripheral polyneuritisInappropriate secretionNeurologic manifestationsSame hospitalizationViral illnessBreast feedIntravenous fluidsCase reportNursing womenNursing mothersAntidiuretic hormoneUnmonitored useTemporal associationPatientsConvulsionsSyndromeWork daysIntoxication
1978
Micropuncture study on the effects of lithium on proximal and distal tubule function in the rat kiney
Hecht B, Kashgarian M, Forrest J, Hayslett J. Micropuncture study on the effects of lithium on proximal and distal tubule function in the rat kiney. Pflügers Archiv - European Journal Of Physiology 1978, 377: 69-74. PMID: 569282, DOI: 10.1007/bf00584376.Peer-Reviewed Original ResearchConceptsFractional reabsorptionDistal tubule functionProximal fractional reabsorptionPlasma lithium concentrationReabsorption of bicarbonateMultiple nephron segmentsEnd-proximal tubuleEffects of lithiumLithium treatmentPotassium secretionAntidiuretic hormoneDistal tubulesMicropuncture studiesTubule functionProximal tubulesDistal convolutionExperimental animalsNephron segmentsReabsorptionDistal portionTubulesPrevious studiesRatsSodiumHormone
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