Inflammation & Homeostasis
Activation of cells of the innate immune system, including neutrophils, monocytes, macrophages, and dendritic cells trigger inflammatory responses that bridge to initiation of adaptive immune responses by lymphocytes. The receptors and signaling molecules that control inflammatory processes, mediate the balance between immune activation and potential for tissue immunopathology, and maintain tissue and organismal homeostasis are under intensive study.
Faculty
Paul B. Beeson Professor of Medicine (Rheumatology) and Professor of Immunobiology; Paul B. Beeson Professor of Medicine, Internal Medicine: Rheumatology; Program Director, Investigative Medicine, Internal Medicine: Rheumatology
Waldemar Von Zedtwitz Professor of Pathology and Professor of Immunobiology; Director, Yale Center for Research on Aging (Y-Age), Pathology
Waldemar Von Zedtwitz Professor of Medicine (Infectious Diseases) and Professor of Epidemiology (Microbial Diseases) and of Microbial Pathogenesis; Affiliated Faculty, Yale Institute for Global Health; Section Chief, Infectious Diseases, Internal Medicine
Sterling Professor of Immunobiology; Investigator, Howard Hughes Medical Institute
Associate Professor of Laboratory Medicine and Immunobiology
Assistant Professor of Medicine (Infectious Diseases)
William S. and Lois Stiles Edgerly Professor of Neurology and Professor of Immunobiology; Chair, Neurology; Neurologist-in-Chief, Yale New Haven Hospital
C.N.H. Long Professor of Immunobiology and of Medicine (Endocrinology)
Associate Professor Term; Director of Graduate Studies, Microbiology PhD Program of Biological and Biomedical Sciences
Sterling Professor of Immunobiology and Professor of Dermatology and of Molecular, Cellular, and Developmental Biology and of Epidemiology (Microbial Diseases); Investigator, Howard Hughes Medical Institute, (HHMI)
Associate Professor of Medicine (Cardiology) and Immunobiology