Nashid H. Chaudhury
Clinical Fellow in Pediatrics (Hospital Medicine)About
Research
Publications
2025
GARLH regulates neuroligin preference for excitatory versus inhibitory synapses
Yamasaki T, Konno K, Krueger-Burg D, Noam Y, Chaudhury N, Morimoto-Tomita M, Salm E, Watanabe M, Brose N, Tomita S. GARLH regulates neuroligin preference for excitatory versus inhibitory synapses. Journal Of Cell Biology 2025, 225: e202507190. PMID: 41329163, DOI: 10.1083/jcb.202507190.Peer-Reviewed Original ResearchConceptsSynaptic specificityInhibitory synapsesNeurotransmitter receptorsGABAA receptorsLocalized to inhibitory synapsesCell adhesion proteinsPostsynaptic neuroliginsTripartite complexNeuroligin isoformsInhibitory GABAA receptorsAdhesion proteinsNeuroliginMolecular mechanismsExcitatory synapsesPostsynaptic sitesReceptorsProteinNL1SynapsesNeurotransmitterIsoformsGABAAMake It Happen NOW: Barriers and Inequities in Eat, Sleep, Console Implementation for Neonatal Opioid Withdrawal Syndrome.
Chaudhury N, Zhao S, Ostfeld-Johns S. Make It Happen NOW: Barriers and Inequities in Eat, Sleep, Console Implementation for Neonatal Opioid Withdrawal Syndrome. Hospital Pediatrics 2025, 15: e99-e101. PMID: 39894048, DOI: 10.1542/hpeds.2025-008332.Peer-Reviewed Original Research
2017
Assembly rules for GABAA receptor complexes in the brain
Martenson JS, Yamasaki T, Chaudhury NH, Albrecht D, Tomita S. Assembly rules for GABAA receptor complexes in the brain. ELife 2017, 6: e30826. PMID: 28816653, PMCID: PMC5577914, DOI: 10.7554/elife.27443.Peer-Reviewed Original Research
2015
Fetal exposure to maternal stress and risk for schizophrenia spectrum disorders among offspring: Differential influences of fetal sex
Fineberg AM, Ellman LM, Schaefer CA, Maxwell SD, Shen L, Chaudhury NH, Cook AL, Bresnahan MA, Susser ES, Brown AS. Fetal exposure to maternal stress and risk for schizophrenia spectrum disorders among offspring: Differential influences of fetal sex. Psychiatry Research 2015, 236: 91-97. PMID: 26753951, PMCID: PMC4767153, DOI: 10.1016/j.psychres.2015.12.026.Peer-Reviewed Original ResearchConceptsSchizophrenia spectrum disordersMaternal stressDaily life stressLife stressLarge birth cohort studyMaternal reportsBirth cohort studySevere life eventsLife eventsCohort studyFetal exposurePregnant womenSpectrum disorderFetal sensitivityPregnancyPregnancy interviewFetal sexMale offspringRiskIndependent ratersSSD casesSex interactionWomenDisordersPresent study
2014
Interneuron precursor transplants in adult hippocampus reverse psychosis-relevant features in a mouse model of hippocampal disinhibition
Gilani AI, Chohan MO, Inan M, Schobel SA, Chaudhury NH, Paskewitz S, Chuhma N, Glickstein S, Merker RJ, Xu Q, Small SA, Anderson SA, Ross ME, Moore H. Interneuron precursor transplants in adult hippocampus reverse psychosis-relevant features in a mouse model of hippocampal disinhibition. Proceedings Of The National Academy Of Sciences Of The United States Of America 2014, 111: 7450-7455. PMID: 24794528, PMCID: PMC4034251, DOI: 10.1073/pnas.1316488111.Peer-Reviewed Original ResearchConceptsHippocampal disinhibitionInterneuron deficitsMouse modelVentral tegmental area dopamine neuron population activityDopamine neuron population activityEmbryonic medial ganglionic eminenceHippocampal metabolic activityHippocampus-dependent cognitionTreatment of schizophreniaDopamine neuron activityNeuron population activityMedial ganglionic eminenceKnockout mouse modelBasal metabolic activityTransplantation of cellsAdult hippocampusProjection neuronsSynaptic inhibitionHippocampal dysfunctionBehavioral hyperresponsivenessInterneuron functionNeuron activityGanglionic eminenceHippocampusMetabolic activity
2013
Imaging Patients with Psychosis and a Mouse Model Establishes a Spreading Pattern of Hippocampal Dysfunction and Implicates Glutamate as a Driver
Schobel SA, Chaudhury NH, Khan UA, Paniagua B, Styner MA, Asllani I, Inbar BP, Corcoran CM, Lieberman JA, Moore H, Small SA. Imaging Patients with Psychosis and a Mouse Model Establishes a Spreading Pattern of Hippocampal Dysfunction and Implicates Glutamate as a Driver. Neuron 2013, 78: 81-93. PMID: 23583108, PMCID: PMC3966570, DOI: 10.1016/j.neuron.2013.02.011.Peer-Reviewed Original ResearchMeSH KeywordsAmino AcidsAnimalsAtrophyBrain MappingBridged Bicyclo Compounds, HeterocyclicDisease Models, AnimalExcitatory Amino Acid AgonistsExcitatory Amino Acid AntagonistsFemaleFollow-Up StudiesFunctional LateralityGlutamic AcidHippocampusHumansImaging, Three-DimensionalKetamineMagnetic Resonance ImagingMaleMiceMice, Inbred C57BLPsychotic DisordersTime FactorsConceptsMouse modelParvalbumin-Expressing InterneuronsAcute ketamineHippocampal hypermetabolismRisk patientsAcute psychosisHippocampal atrophyHippocampal metabolismPathogenic driverHippocampal dysfunctionExtracellular glutamatePsychosis onsetHypermetabolismPsychotic disordersBasal stateMRI toolsPatientsAtrophyPsychosisHippocampusKetamineGlutamateAbnormalitiesSimilar regional patternLY379268