activity PTH-dependent stabilization of RANKL mRNA is associated with a selective increase in binding of phosphorylated KSRP, an AU-rich element binding-protein, to RANKL transcripts
activity Parathyroid Hormone does not change the expression of adenylate-uridylate-rich element binding proteins as part of the mechanism by which it stabilizes Receptor Activator of NF Kappa B Ligand Transcripts
activity The anabolic actions of PTH are mediated in part through a Colony Stimulating Factor 1-sphingosine-1-phosphate paracrine loop
activity The anabolic actions of PTH are mediated in part through a Colony Stimulating Factor 1-sphingosine-1-phosphate paracrine loop
activity Selective deletion of the Soluble Colony Stimulating Factor 1 isoform in vivo eliminates estrogen-deficiency bone loss in mice
activity Selective knock out the membrane-bound Colony Stimulating Factor-1 (CSF1) results in an increased anabolic response to PTH in mice
activity Selective deletion of the membrane-bound Colony Stimulating Factor 1 isoform in vivo does not affect estrogen-deficiency bone loss in mice
activity Selective deletion of the membrane-bound Colony Stimulating Factor-1 (CSF1) isoform results in increased bone density and hypertriglyceridemia in mice
activity Overexpression of Dkk-1 in osteoblasts reduced bone mass but unaltered anabolic response to PTH in mice
activity Selective Expression of the Soluble or Membrane-Bound Colony Stimulating Factor-1 (CSF-1) Isoform in Osteoblasts Accelerates Estrogen-Deficiency Bone Loss
activity Constitutive Activation of Wnt signaling through Low-Density Lipoprotein-Related Protein-5 Elevates Expression of TGF-beta, Fibronectin and Osteocalcin in Primary Murine Osteoblasts.
activity Over-expression of the cell-surface isoform of Colony Stimulating Factor-1 in osteoblasts causes increased osteoclastogenesis and bone loss
honor Featured Investigator, Recipient of a Pilot and Feasibility Project Award
activity Cell-Surface CSF-1 Expression by Osteoblasts Supports Osteoclastogenesis
activity Inhibition of Epstein-Barr virus replication by a novel L-nucleoside, 2'-fluoro-5-methyl-beta-L-arabinofuranosyluracil.
activity Potent inhibition of Epstein-Barr virus by phosphorothioate oligodeoxynucleotides without sequence specification.
activity Identification of High and low Epstein-Barr virus producing HR-1 cells
honor Junior Investigator Award
honor Phi Beta Kappa