2008
Nicotine decreases DNA methyltransferase 1 expression and glutamic acid decarboxylase 67 promoter methylation in GABAergic interneurons
Satta R, Maloku E, Zhubi A, Pibiri F, Hajos M, Costa E, Guidotti A. Nicotine decreases DNA methyltransferase 1 expression and glutamic acid decarboxylase 67 promoter methylation in GABAergic interneurons. Proceedings Of The National Academy Of Sciences Of The United States Of America 2008, 105: 16356-16361. PMID: 18852456, PMCID: PMC2570996, DOI: 10.1073/pnas.0808699105.Peer-Reviewed Original ResearchMeSH KeywordsAnimalsDNA (Cytosine-5-)-Methyltransferase 1DNA (Cytosine-5-)-MethyltransferasesDNA MethylationDown-Regulationgamma-Aminobutyric AcidGene Expression Regulation, EnzymologicGlutamate DecarboxylaseHippocampusMaleMiceNeuronsNicotinePromoter Regions, GeneticReceptors, CholinergicReelin ProteinRNA, MessengerTime FactorsUp-RegulationConceptsGABAergic neuronsFrontal cortexGABAergic interneuronsSchizophrenia patientsMedium spiny GABAergic neuronsDNMT1 expressionNicotinic receptor populationsStriatal GABAergic neuronsBlood-brain barrierHippocampal GABAergic interneuronsNicotine-induced decreasesDifferent cortical layersMouse frontal cortexDNA methyltransferase 1 expressionDNMT1 mRNA expressionSmoking habitsTobacco smokingReceptor dysfunctionMuscarinic receptorsSame dosesReceptor populationMethyltransferase 1 expressionCortical layersMRNA expressionHypermethylation of promoters
2007
Angiotensin‐(1–7) prevents cardiac remodeling during angiotensin II‐induced hypertension
Grobe J, Mecca A, Lingis M, Shenoy V, Bolton T, Machado J, Speth R, Raizada M, Katovich M. Angiotensin‐(1–7) prevents cardiac remodeling during angiotensin II‐induced hypertension. The FASEB Journal 2007, 21: a896-a897. DOI: 10.1096/fasebj.21.6.a896-d.Peer-Reviewed Original ResearchAng IICardiac remodelingBlood pressureInterstitial fibrosisMyocyte hypertrophyMale Sprague-Dawley ratsAng II infusionAnti-remodeling effectsEnd-organ damageRenin-angiotensin systemSprague-Dawley ratsII infusionAng receptorsAngiotensin receptorsAngiotensin IIMyocardial fibrosisRat modelMyocyte diameterEnzyme 2Receptor populationAngHypertensionFibrosisSignificant attenuationLentiviral delivery
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