2016
Recurrent genetic defects in classical Hodgkin lymphoma cell lines
Hudnall SD, Meng H, Lozovatsky L, Li P, Strout M, Kleinstein SH. Recurrent genetic defects in classical Hodgkin lymphoma cell lines. Leukemia & Lymphoma 2016, 57: 2890-2900. PMID: 27121023, DOI: 10.1080/10428194.2016.1177179.Peer-Reviewed Original ResearchConceptsMitosis-related genesSingle nucleotide variantsCHL cell linesCell linesRecurrent genetic defectsPathogenic single nucleotide variantsHL cell linesMitotic genesChromosome duplicationClassical Hodgkin lymphoma cell linesGenomic instabilityGenetic analysisWhole-exome sequencingNucleotide variantsGenesHodgkin's lymphoma cell linesLymphoma cell linesNumber variantsKaryotypic analysisGenetic defectsWealth of informationPoor growthVariantsDuplicationLines
2012
Human herpesvirus 6 positive Reed–Sternberg cells in nodular sclerosis Hodgkin lymphoma
Siddon A, Lozovatsky L, Mohamed A, Hudnall SD. Human herpesvirus 6 positive Reed–Sternberg cells in nodular sclerosis Hodgkin lymphoma. British Journal Of Haematology 2012, 158: 635-643. PMID: 22757777, DOI: 10.1111/j.1365-2141.2012.09206.x.Peer-Reviewed Original ResearchMeSH KeywordsAdolescentAdultAge FactorsAgedBlotting, WesternCell Line, TumorDNA, ViralEpstein-Barr Virus InfectionsFemaleHerpesvirus 4, HumanHerpesvirus 6, HumanHodgkin DiseaseHumansImmunohistochemistryLymphoma, FollicularMaleMiddle AgedPolymerase Chain ReactionReed-Sternberg CellsRoseolovirus InfectionsSclerosisYoung AdultConceptsNodular sclerosis Hodgkin lymphomaNodular sclerosis HLHHV-6 DNAEpstein-Barr virusHHV-6Hodgkin's lymphomaReed-Sternberg cellsPolymerase chain reactionRS cellsNSHL casesEBV-negative diseaseReactive lymphoid hyperplasiaClassical Hodgkin lymphomaBi-modal age distributionHHV-6 genomeYounger patientsInfectious etiologyLymphoid hyperplasiaPCR positivityYounger ageImmunohistochemistryMost casesConflicting resultsChain reactionCD30
2007
Comparative flow immunophenotypic features of the inflammatory infiltrates of Hodgkin lymphoma and lymphoid hyperplasia
Hudnall SD, Betancourt E, Barnhart E, Patel J. Comparative flow immunophenotypic features of the inflammatory infiltrates of Hodgkin lymphoma and lymphoid hyperplasia. Cytometry Part B Clinical Cytometry 2007, 74B: 1-8. PMID: 18061945, DOI: 10.1002/cyto.b.20376.Peer-Reviewed Original ResearchConceptsRegulatory T cellsMixed cellularity HLReactive lymphoid hyperplasiaB cell ratioHodgkin's lymphomaReactive lymphocytic infiltrateT cellsLymphoid hyperplasiaInflammatory infiltrateB cellsCD8 ratioNKT cellsLymphocytic infiltrateHL subtypesLymph nodesImmunophenotypic featuresCell ratioMixed cellularity Hodgkin's lymphomaMalignant Reed-Sternberg cellsEBV-negative casesLymphocyte depletion subtypeRich inflammatory infiltratePercentage of CD4Fluorochrome-conjugated monoclonal antibodiesReed-Sternberg cells
2001
Composite nodular lymphocyte-predominance Hodgkin disease and gamma-heavy-chain disease: a case report and review of the literature.
Hudnall S, Alperin J, Petersen J. Composite nodular lymphocyte-predominance Hodgkin disease and gamma-heavy-chain disease: a case report and review of the literature. Archives Of Pathology & Laboratory Medicine 2001, 125: 803-7. PMID: 11371236, DOI: 10.5858/2001-125-0803-cnlphd.Peer-Reviewed Original ResearchConceptsNodular lymphocyte predominance Hodgkin's diseaseLymphocyte predominance Hodgkin's diseaseHodgkin's diseaseChain diseaseAutoimmune hemolytic anemiaB-cell natureB-cell lymphoproliferative processesComplete remissionGraves' diseaseLymph nodesCase reportLymphoproliferative processMonoclonal gammopathyHemolytic anemiaBone marrowUnique caseDiseaseClonal relationshipFurther treatment