2025
Multiphysics Simulations of a Bioprinted Pulsatile Fontan Conduit.
Hu Z, Herrmann J, Schwarz E, Gerosa F, Emuna N, Humphrey J, Feinberg A, Hsia T, Skylar-Scott M, Marsden A. Multiphysics Simulations of a Bioprinted Pulsatile Fontan Conduit. Journal Of Biomechanical Engineering 2025, 147 PMID: 40172060, DOI: 10.1115/1.4068319.Peer-Reviewed Original ResearchConceptsSuperior vena cavaInferior vena cavaIncreased cardiac outputCardiac outputVena cavaElevated central venous pressureConduit performanceCongenital heart patientsCentral venous pressureDiminished cardiac outputFluid-structure interactionMultiphysics computational frameworkFontan surgeryFontan circulationFontan physiologyFontan conduitPalliative procedureLumped parameter networkPulmonary arteryCardiomyocyte contractilityValve placementMyocardial contractilityVenous pressureFontanVenous circulationTakotsubo Syndrome in a 47-Year-Old Woman With Repaired Tetralogy of Fallot
Kayani J, Bailey L, Hopkins K, Zaidi A, Love B. Takotsubo Syndrome in a 47-Year-Old Woman With Repaired Tetralogy of Fallot. JACC Case Reports 2025, 30: 102804. PMID: 39886417, PMCID: PMC11775812, DOI: 10.1016/j.jaccas.2024.102804.Peer-Reviewed Original ResearchCase of takotsubo syndromeCongenital heart diseaseTakotsubo syndromePremenopausal womenAdult congenital heart diseaseCongenital heart disease populationComplex congenital heart diseaseEvidence of coronary artery diseaseHeart diseaseHeart disease populationExtremely rare caseAcquired cardiac diseaseRegional systolic dysfunctionCoronary artery diseaseAcute plaque ruptureRepaired tetralogySystolic dysfunctionNonischemic cardiomyopathyBroken-heart syndromeRare formAcute onsetMyocardial contractilityArtery diseaseCardiac diseaseChest tightness
2019
The effect of cardiac shock wave therapy on myocardial function and perfusion in the randomized, triple-blind, sham-procedure controlled study
Čelutkienė J, Burneikaitė G, Shkolnik E, Jakutis G, Vajauskas D, Čerlinskaitė K, Zuozienė G, Petrauskienė B, Puronaitė R, Komiagienė R, Butkuvienė I, Steponėnienė R, Misiūra J, Laucevičius A. The effect of cardiac shock wave therapy on myocardial function and perfusion in the randomized, triple-blind, sham-procedure controlled study. Cardiovascular Ultrasound 2019, 17: 13. PMID: 31272465, PMCID: PMC6610956, DOI: 10.1186/s12947-019-0163-1.Peer-Reviewed Original ResearchConceptsCardiac shock wave therapyDobutamine stress echocardiographyWall motion score indexSingle photon emission computed tomographyEffect of cardiac shock wave therapySham procedure groupMonths follow-upShock wave therapyStudy groupPerfusion defectsTriple-blindSPECT resultsProcedure groupCSWT treatmentRegional myocardial contractilityMyocardial contractilityMyocardial functionWave therapyMyocardial ischemiaCardiac single photon emission computed tomographyGroups compared to baselineStress-induced myocardial ischemiaPhoton emission computed tomographyCo-primary endpointsWall motion abnormalities24 Cardiovascular Physiology Integrative Function
Crystal G, Assaad S, Heerdt P, Desso R, Swenson J, Kolbay P, Kuck K, Decou J. 24 Cardiovascular Physiology Integrative Function. 2019, 473-519. DOI: 10.1016/b978-0-323-48110-6.00024-7.ChaptersBlood flowPerioperative cardiovascular monitoringEnd-systolic pressure-volume relationPulmonary artery catheterMyocardial oxygen demandCoronary blood flowSufficient blood flowCardiac function curveTissue blood flowTissue oxygen deliveryPressure-volume loopsTissue oxygen consumptionCirculatory reflexesPerioperative periodArtery catheterBody tissuesEjection fractionPressure-volume relationCardiac outputCardiac functionMyocardial contractilityMyocardial ischemiaStroke volumePatient managementOxygen delivery
2012
miR-1 mediated suppression of Sorcin regulates myocardial contractility through modulation of Ca2+ signaling
Ali R, Huang Y, Maher SE, Kim RW, Giordano FJ, Tellides G, Geirsson A. miR-1 mediated suppression of Sorcin regulates myocardial contractility through modulation of Ca2+ signaling. Journal Of Molecular And Cellular Cardiology 2012, 52: 1027-1037. PMID: 22326846, DOI: 10.1016/j.yjmcc.2012.01.020.Peer-Reviewed Original ResearchMeSH KeywordsAnimalsBase SequenceCalcium SignalingCalcium-Binding ProteinsCardiac VolumeCardiomyopathiesCell LineDEAD-box RNA HelicasesHeartHumansMaleMiceMice, 129 StrainMice, Inbred C57BLMice, KnockoutMicroRNAsMyocardial ContractionMyocardiumRibonuclease IIIRNA InterferenceRNA, Small InterferingUp-RegulationConceptsCardiac functionMiR-1Normal cardiac contractile functionEnd-stage cardiomyopathyCardiac contractile functionWild-type miceCalcium signalingExcitation-contraction couplingModulation of Ca2Cultured mouse cardiomyocytesAcute cardiomyopathyMiR-1 targetsHeart failureMyocardial contractilityMiR-1 knockdownContractile functionAntagomir treatmentSorcin expressionCalcium homeostasisKnockdown miceSorcin levelsCardiac phenotypeMouse cardiomyocytesCritical mediatorPathological relevance
2003
Cardiac Resynchronization: A Brief Synopsis Part I: Patient Selection and Results from Clinical Trials
Kalinchak DM, Schoenfeld MH. Cardiac Resynchronization: A Brief Synopsis Part I: Patient Selection and Results from Clinical Trials. Journal Of Interventional Cardiac Electrophysiology 2003, 9: 155-161. PMID: 14574027, DOI: 10.1023/a:1026276205618.Peer-Reviewed Original ResearchConceptsHeart failureBiventricular pacingClass IV heart failureAcute hemodynamic benefitAcute hemodynamic parametersDepressed ejection fractionNYHA class IIISevere heart failureBundle branch blockLong-term functional capacityVentricular dysynchronyPrevious therapyVascular resistanceHemodynamic benefitsEjection fractionICD placementPatient selectionBranch blockMyocardial contractilityClinical trialsMechanical dysfunctionHemodynamic parametersMild symptomsSeparate indicationsFunctional capacity
2001
Linkage of Left Ventricular Contractility to Chromosome 11 in Humans
Arnett D, Devereux R, Kitzman D, Oberman A, Hopkins P, Atwood L, Dewan A, Rao DC. Linkage of Left Ventricular Contractility to Chromosome 11 in Humans. Hypertension 2001, 38: 767-772. PMID: 11641284, DOI: 10.1161/hy1001.092650.Peer-Reviewed Original ResearchMeSH KeywordsBlack PeopleChromosome MappingChromosomes, Human, Pair 11Chromosomes, Human, Pair 12Chromosomes, Human, Pair 22EchocardiographyFemaleGenetic LinkageGenome, HumanGenotypeHeart VentriclesHumansHypertensionLod ScoreMaleMicrosatellite RepeatsMiddle AgedMyocardial ContractionPhenotypeQuantitative Trait, HeritableWhite PeopleConceptsCongestive heart failureLV contractilityHeart failureVentricular contractilityMyocardial contractilityImpaired LV contractilityLeft ventricular contractilityAge 60 yearsHypertension Genetic Epidemiology NetworkGenetic Epidemiology NetworkBiethnic sampleCardiovascular deathContractile impairmentFamilial hypertrophic cardiomyopathyMean ageEchocardiographic measurementsHypertrophic cardiomyopathyHypertensive siblingsContractilityEpidemiology NetworkMajor causeSusceptibility genesAgeEthnic groupsSuggestive evidence
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