2020
Epidemiology, Pathophysiology, and Genetics of Primary Hyperparathyroidism
Minisola S, Arnold A, Belaya Z, Brandi ML, Clarke BL, Hannan FM, Hofbauer LC, Insogna KL, Lacroix A, Liberman U, Palermo A, Pepe J, Rizzoli R, Wermers R, Thakker RV. Epidemiology, Pathophysiology, and Genetics of Primary Hyperparathyroidism. Journal Of Bone And Mineral Research 2020, 37: 2315-2329. PMID: 36245271, PMCID: PMC10092691, DOI: 10.1002/jbmr.4665.Peer-Reviewed Original ResearchConceptsPrimary hyperparathyroidismFamilial hypocalciuric hypercalcemiaMultiple endocrine neoplasia syndromeCalcium-containing kidney stonesIntestinal calcium hyperabsorptionMore parathyroid glandsParathyroid hormone levelsAsymptomatic primary hyperparathyroidismCalcium-sensing receptorEndocrine neoplasia syndromeNeonatal severe hyperparathyroidismHyperparathyroidism-jaw tumor syndromeJaw tumor syndromeCalcium hyperabsorptionPostmenopausal womenSevere hyperparathyroidismParathyroid hormoneSerum calciumHypocalciuric hypercalcemiaPathophysiologic basisParathyroid cellsParathyroid glandsHormone levelsFiltered loadMetabolic factors
2010
Low urinary citrate: an overview.
Zacchia M, Preisig P. Low urinary citrate: an overview. Journal Of Nephrology 2010, 23 Suppl 16: s49-56. PMID: 21170889.Peer-Reviewed Original ResearchConceptsUrinary citrate excretionCitrate excretionStone formationCalcium stone formationProximal tubule cellsAcid-base statusKidney stone formationRisk factorsFiltered loadImportant physiological determinantGlomerular filtrateTubule cellsAcid loadExcretionAlkaline loadCitrate levelsHypocitraturiaMetabolic processesMitochondrial metabolic processesPhysiological determinantsMetabolismMitochondrial metabolismCitrate metabolismNaDC1The calcium-sensing receptor.
Geibel JP. The calcium-sensing receptor. Journal Of Nephrology 2010, 23 Suppl 16: s130-5. PMID: 21170870.Peer-Reviewed Original ResearchConceptsCalcium-sensing receptorExtracellular environmentReceptor-targeted drugsThick ascending limbFiltered loadAscending limbNephron segmentsAmino acidsHandling of waterReceptorsImportant roleBasic physiologyImportant memberSalt absorptionRecent studiesDivalent ion concentrationSalinityCalciumIonic absorptionCaSR
1994
Acute Regulation of Na+:HCO3- Cotransport System In Kidney Proximal Tubules
Soleimani M, Bizal G, Hattabaugh Y, Aronson P, Bergman J. Acute Regulation of Na+:HCO3- Cotransport System In Kidney Proximal Tubules. NATO ASI Series 1994, 309-317. DOI: 10.1007/978-3-642-79301-1_36.ChaptersKidney proximal tubulesProximal tubulesKidney proximal tubule cellsProximal tubule cellsCotransport systemFiltered loadBlood pHHCO3- reabsorptionTubule cellsAcute regulatory mechanismsAcute regulationTransport of HCO3N-ethylmaleimideTubulesBasolateral membraneRecent studiesCotransporterLumenTransportersFunctional characteristicsCellsRegulatory mechanismsNumerous typesBloodSecretion
1984
Renal hydrolysis of absorbed protein: influence of load and lysosomal pH
Camargo MJ, Sumpio BE, Maack T. Renal hydrolysis of absorbed protein: influence of load and lysosomal pH. American Journal Of Physiology 1984, 247: f656-f664. PMID: 6093557, DOI: 10.1152/ajprenal.1984.247.4.f656.Peer-Reviewed Original ResearchConceptsNormal filtered loadEfflux of radioactivityRenal tubular cellsEffect of alkalinizationProtein absorption dropletsRenal catabolismNormal lysosomal functionLysosomotropic weak basesFiltered loadTubular cellsKidney 1Abnormal depositionRat kidneyKidneyAbsorption dropletsSaturable processIntracellular hydrolysisSaturable functionLysosomal functionPerfusateLysosomal pHLow uptake rateSephadex chromatographyReversible mannerHalf times
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