2020
Residual endotoxin induces primary graft dysfunction through ischemia-reperfusion-primed alveolar macrophages
Akbarpour M, Lecuona E, Chiu S, Wu Q, Querrey M, Fernandez R, Nunez-Santana F, Sun H, Ravi S, Kurihara C, Walter JM, Joshi N, Ren Z, Roberts SC, Hauser A, Kreisel D, Li W, Chandel N, Misharin AV, Mohanakumar T, Budinger GRS, Bharat A. Residual endotoxin induces primary graft dysfunction through ischemia-reperfusion-primed alveolar macrophages. Journal Of Clinical Investigation 2020, 130: 4456-4469. PMID: 32692317, PMCID: PMC7410086, DOI: 10.1172/jci135838.Peer-Reviewed Original ResearchConceptsTissue-resident alveolar macrophagesPrimary graft dysfunctionHuman donor lungsNeutrophil recruitmentGraft dysfunctionDonor lungsBacterial pneumoniaAlveolar macrophagesBone marrow chimeric miceDonor lung injuryActivation of TLR4Blockade of TLR4MyD88-dependent mannerResidual endotoxinCoreceptor CD14Lung transplantationLung injuryNeutrophil infiltrationFatal syndromeSuccessful treatmentMouse modelChimeric miceBacterial endotoxinTLR4Endotoxin
1986
Progressive Epstein-Barr Virus Infection
Andiman W, Katz B, Miller G. Progressive Epstein-Barr Virus Infection. 1986, 276-286. DOI: 10.1007/978-1-4612-4958-0_33.Peer-Reviewed Original ResearchEpstein-Barr virusInfectious mononucleosisEpstein-Barr virus infectionDisorders of immunitySpectrum of diseaseEBV infectionImmunologic controlFatal syndromeVirus infectionClinical conditionsNosologic entityB cellsViral replicationInfectionMononucleosisDiseaseAbnormal degreeUnchecked proliferationImmunopathogenesisRelapsingNeoplasiaSyndromeAntigenImmunity
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