2023
Body Cavity Fluid Cytology
Wang M. Body Cavity Fluid Cytology. 2023, 57-71. DOI: 10.1007/978-3-031-44289-6_5.ChaptersSerous effusionsMinimally invasive procedureExcessive accumulation of fluidAspiration of fluidAccumulation of fluidLymphocytic effusionsBenign causesTuberculous effusionsNeedle aspirationSystemic lupusIdentifying malignancyChylous effusionInvasive proceduresPeritoneal cavityCytologic evaluationMesothelial cellsEffusionSerous fluidInflammatory effusionsEosinophilic effusions
2022
Ascites and its complications
Garcia‐Tsao G. Ascites and its complications. 2022, 522-531. DOI: 10.1002/9781119600527.ch59.Peer-Reviewed Reviews, Practice Guidelines, Standards, and Consensus StatementsCirrhotic ascitesPresence of ascitesMainstay of therapySevere liver diseaseSpontaneous bacterial peritonitisAccumulation of fluidHepatorenal syndromePeritoneal malignancyUncomplicated ascitesAbdominal ultrasonographySodium restrictionCirrhotic patientsHepatic hydrothoraxBacterial peritonitisHeart failureLiver diseaseDecompensated stageCommon causePeritoneal cavityAscitesPatientsMost cost-effective methodBacterial infectionsCirrhosisCommon typeAscites and its complications
García‐Tsao G. Ascites and its complications. 2022, 2024-2043. DOI: 10.1002/9781119600206.ch98.Peer-Reviewed Original ResearchHepatic sinusoidal pressureHepatic venous pressure gradientSerum-ascites albumin gradientAcute kidney injuryPresence of ascitesVenous pressure gradientMain pathogenic mechanismPredominance of neutrophilsSinusoidal pressureAccumulation of fluidAbdominal ultrasonographyKidney injurySodium restrictionCirrhotic patientsPortal pressureSodium retentionDecompensated stageInflammatory processPolymorphonuclear cellsPeritoneal cavityAscites albuminAscitic fluidAscitesCirrhosisPathogenic mechanisms
2016
Approach to the Patient with Ascites and Its Complications
Garcia‐Tsao G. Approach to the Patient with Ascites and Its Complications. 2016, 447-458. DOI: 10.1002/9781118512104.ch55.Peer-Reviewed Reviews, Practice Guidelines, Standards, and Consensus StatementsLarge-volume paracentesisSpontaneous bacterial peritonitisNew-onset ascitesPresence of ascitesAcute renal failureFirst-line therapyMainstay of therapySerial large-volume paracentesisEtiology of ascitesAccumulation of fluidHepatorenal syndromePeritoneal malignancyAbdominal ultrasonographyRefractory ascitesSodium restrictionHepatic hydrothoraxLine therapyRenal failureBacterial peritonitisHeart failureDecompensated stageCirrhotic ascitesCommon causePeritoneal cavityAscites
2015
Ascites and Its Complications
Garcia‐Tsao G. Ascites and Its Complications. 2015, 2087-2106. DOI: 10.1002/9781118512074.ch106.Peer-Reviewed Reviews, Practice Guidelines, Standards, and Consensus StatementsAcute kidney injuryHepatorenal syndromeCompatible clinical presentationPostliver transplant outcomesCause of ascitesPancreatic ductal systemAccumulation of fluidKidney injuryTransplant outcomesTuberculous peritonitisHeart failureClinical presentationPancreatic ascitesDecompensated stageMalignant ascitesCirrhosisPeritoneal cavityAscitesBetter survivalDuctal systemPatientsPancreatic fluidOptimal managementComplicationsMain cause
2012
Ascites
Garcia‐Tsao G. Ascites. 2012, 103-106. DOI: 10.1002/9781118321386.ch17.Peer-Reviewed Reviews, Practice Guidelines, Standards, and Consensus StatementsRight heart failurePeritoneal malignancyHeart failureHepatic sinusoidal pressureLow-protein ascitesNew-onset ascitesSpontaneous bacterial peritonitisEtiology of ascitesRoutine laboratory testsRisk of infectionAccumulation of fluidDiagnostic paracentesisBacterial peritonitisCareful historyPhysical examinationCirrhotic ascitesLiver architecturePeritoneal cavityHepatic sinusoidsAscitesPeritoneal lymphaticsCell countCirrhosisProtein levelsSinusoidal pressure
2010
Drosophila glia use a conserved cotransporter mechanism to regulate extracellular volume
Leiserson WM, Forbush B, Keshishian H. Drosophila glia use a conserved cotransporter mechanism to regulate extracellular volume. Glia 2010, 59: 320-332. PMID: 21125654, PMCID: PMC3005002, DOI: 10.1002/glia.21103.Peer-Reviewed Original ResearchMeSH KeywordsAction PotentialsAnimalsAnimals, Genetically ModifiedBlood-Nerve BarrierCells, CulturedDrosophilaDrosophila ProteinsExtracellular SpaceHumansIn Vitro TechniquesLarvaMicroscopy, Electron, TransmissionModels, BiologicalMutationNeural ConductionNeurogliaNeuronsPeripheral NervesProtein Serine-Threonine KinasesSymportersTwo-Hybrid System TechniquesConceptsHuman NKCC1Yeast two hybrid assaysExtracellular solute compositionLarvae mutantDrosophila gliaNcc69Osmotic homeostasisExtracellular volumeMolecular mechanismsNervous systemOrthologsExtracellular solutesPhysiological mechanismsBlood-brain barrierBlood-nerve barrierSimilar roleAccumulation of fluidAction potential conductionGlial cellsPeripheral neuropathyNKCC1Serious health threatDetectable impactBlood barrierGlia
2005
Ascites
Kbalid S, Garcia-Tsao G. Ascites. Clinical Gastroenterology 2005, 285-299. DOI: 10.1007/978-1-59259-885-4_19.Peer-Reviewed Reviews, Practice Guidelines, Standards, and Consensus StatementsSevere ovarian hyperstimulation syndromeOvarian hyperstimulation syndromeCause of ascitesAccumulation of fluidHyperstimulation syndromePeritoneal malignancyCompensated cirrhosisPeritoneal tuberculosisPortal hypertensionCommon complicationCardiac failureProspective studyCommon causeDifferential diagnosisPeritoneal cavityAscitesCirrhosisCumulative probabilityPatientsCauseHypertensionComplicationsMalignancySyndromeEtiology
2001
Management of Brain Edema Complicating Stroke
Rosand J, Schwamm L. Management of Brain Edema Complicating Stroke. Journal Of Intensive Care Medicine 2001, 16: 128-141. DOI: 10.1046/j.1525-1489.2001.00128.x.Peer-Reviewed Original ResearchCerebral blood flowSecondary brain injuryBrain edemaMagnetic resonance imagingBrain parenchymaVasogenic edemaBrain injuryBlood-brain barrier integrityGlobal cerebral blood flowIschemic brain swellingLocal tissue dysfunctionSerial Neurologic AssessmentIschemic brain edemaOnset of symptomsIntracranial blood volumeIschemic cell injuryResult of injurySpecific MRI sequencesAccumulation of fluidDiffusion-weighted imagingRheologic agentsNeurologic deteriorationBrain swellingDecompressive surgeryIschemic strokeManagement of Brain Edema Complicating Stroke
Rosand J, Schwamm L. Management of Brain Edema Complicating Stroke. Journal Of Intensive Care Medicine 2001, 16: 128-141. DOI: 10.1177/088506660101600303.Peer-Reviewed Original ResearchCerebral blood flowSecondary brain injuryBrain edemaMagnetic resonance imagingBrain parenchymaVasogenic edemaBrain injuryBlood-brain barrier integrityGlobal cerebral blood flowIschemic brain swellingLocal tissue dysfunctionSerial Neurologic AssessmentIschemic brain edemaOnset of symptomsIntracranial blood volumeIschemic cell injuryResult of injurySpecific MRI sequencesAccumulation of fluidDiffusion-weighted imagingRheologic agentsNeurologic deteriorationBrain swellingDecompressive surgeryIschemic stroke
1995
Cirrhotic ascites: pathogenesis and management.
Garcia-Tsao G. Cirrhotic ascites: pathogenesis and management. The Gastroenterologist 1995, 3: 41-54. PMID: 7743121.BooksConceptsPathogenesis of ascitesComplications of cirrhosisNegative sodium balanceAccumulation of fluidSinusoidal hypertensionLiver transplantationSalt restrictionPortosystemic shuntingPeripheral vasoconstrictorNonsurgical sideIntravascular volumeCurrent therapiesIntraperitoneal fluidSodium balanceTherapeutic priorityTherapeutic measuresPeritoneal spacePeritoneal cavitySystemic circulationAscitesEfflux of fluidPatientsDiureticsPathogenesisFuture studies
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