2019
Gastrointestinal Tract
Abi-Raad R, Cai G. Gastrointestinal Tract. 2019, 365-384. DOI: 10.1007/978-3-030-21799-0_13.ChaptersGastrointestinal tract lesionsTract lesionsUltrasound-guided fine-needle aspiration cytologyEndoscopic ultrasound-guided fine-needle aspiration cytologyFine needle aspiration cytologyGastrointestinal stromal tumorsKey cytologic featuresNeedle aspiration cytologySubmucosal lesionsStromal tumorsBarrett's esophagusAppropriate triageGastric adenocarcinomaAncillary studiesDifferential diagnosisAspiration cytologyCytologic featuresEUS-FNAGastrointestinal tractAspiration materialIntestinal tractPreliminary diagnosisLesionsDiagnosisImmediate evaluation
2016
Reorganization of Intact Descending Motor Circuits to Replace Lost Connections After Injury
Fink KL, Cafferty WB. Reorganization of Intact Descending Motor Circuits to Replace Lost Connections After Injury. Neurotherapeutics 2016, 13: 370-381. PMID: 26846379, PMCID: PMC4824020, DOI: 10.1007/s13311-016-0422-x.Peer-Reviewed Original ResearchConceptsSpinal cord injuryCentral nervous systemMotor pathwaysFunctional recoveryMotor functionMotor circuitsIntact circuitsIncomplete spinal cord injuryPartial spinal cord injuryAdult central nervous systemCorticospinal tract lesionsLimited spontaneous recoveryPermanent functional impairmentSpontaneous functional recoveryExperimental rodent modelsIntrinsic growth capacityRestoration of functionFine motor behaviorRaphespinal tractsDenervated sideTract lesionsCord injuryRubrospinal tractReticulospinal tractCNS neurons
2015
Plasticity of Intact Rubral Projections Mediates Spontaneous Recovery of Function after Corticospinal Tract Injury
Siegel CS, Fink KL, Strittmatter SM, Cafferty WB. Plasticity of Intact Rubral Projections Mediates Spontaneous Recovery of Function after Corticospinal Tract Injury. Journal Of Neuroscience 2015, 35: 1443-1457. PMID: 25632122, PMCID: PMC4308593, DOI: 10.1523/jneurosci.3713-14.2015.Peer-Reviewed Original ResearchMeSH KeywordsAnimalsDesigner DrugsFunctional LateralityGene Expression RegulationGlial Fibrillary Acidic ProteinLocomotionMaleMiceMice, Inbred C57BLMice, TransgenicMuscle StrengthMyelin ProteinsNeuronal PlasticityNogo ProteinsPsychomotor DisordersPyramidal TractsRaphe NucleiRecovery of FunctionSpinal Cord InjuriesStereotyped BehaviorTime FactorsConceptsSpinal cord injurySpontaneous functional recoveryFunctional recoverySpontaneous recoveryIncomplete spinal cord injuryCorticospinal tract lesionsWeeks of lesionCorticospinal tract injuryNogo receptor 1Nucleus raphe magnusTract injuryRubrospinal projectionsTract lesionsCord injuryRaphe magnusCircuit rearrangementsAdult CNSCircuit plasticityLocomotor functionAdult micePharmacogenetic toolsRed nucleusRubral projectionReceptor 1Extensive sprouting
2012
1171 Significance of Incidental Gastrointestinal (GI) Tract Lesions Identified by Positron Emission Tomography (PET)
Singerman J, Tian J, Nagar A. 1171 Significance of Incidental Gastrointestinal (GI) Tract Lesions Identified by Positron Emission Tomography (PET). Gastrointestinal Endoscopy 2012, 75: ab179. DOI: 10.1016/j.gie.2012.04.163.Peer-Reviewed Original Research
2003
Rho Kinase Inhibition Enhances Axonal Regeneration in the Injured CNS
Fournier AE, Takizawa BT, Strittmatter SM. Rho Kinase Inhibition Enhances Axonal Regeneration in the Injured CNS. Journal Of Neuroscience 2003, 23: 1416-1423. PMID: 12598630, PMCID: PMC6742251, DOI: 10.1523/jneurosci.23-04-01416.2003.Peer-Reviewed Original ResearchMeSH KeywordsADP Ribose TransferasesAmidesAnimalsAxonsBotulinum ToxinsCells, CulturedChick EmbryoEnzyme InhibitorsFemaleGanglia, SpinalIntracellular Signaling Peptides and ProteinsMotor ActivityMyelin ProteinsNerve RegenerationNeuritesNogo ProteinsPC12 CellsProtein Serine-Threonine KinasesPyridinesRatsRats, Sprague-Dawleyrho GTP-Binding Proteinsrho-Associated KinasesSpinal Cord InjuriesConceptsAxonal regenerationAdult ratsNeurite outgrowthCorticospinal tract lesionsNeurite outgrowth inhibitorChick DRG neuronsRho-kinase inhibitionCST fibersDRG neuronsCST lesionLocomotor recoveryTract lesionsSpinal cordOutgrowth inhibitorInhibits neurite outgrowthNogo-66Activity levelsMyelinKinase inhibitionLesionsActivation of RhoRatsC3 transferaseInhibition of p160ROCKInhibitors
2000
Opiate receptor avidity is increased in rhesus monkeys following unilateral optic tract lesion combined with transections of corpus callosum and hippocampal and anterior commissures
Cohen R, Carson R, Saunders R, Doudet D. Opiate receptor avidity is increased in rhesus monkeys following unilateral optic tract lesion combined with transections of corpus callosum and hippocampal and anterior commissures. Brain Research 2000, 879: 1-6. PMID: 11010998, DOI: 10.1016/s0006-8993(00)02528-2.Peer-Reviewed Original ResearchConceptsOpiate receptor avidityPositron emission tomographyReceptor avidityAnterior commissureCorpus callosumKappa-opiate receptor antagonistUnilateral optic tract lesionRhesus monkeysOptic tract lesionsBrain functional activitySplit animalsIpsilateral changesOptic tractTract lesionsUnilateral lesioningPosterior putamenReceptor antagonistHealthy monkeysLateral cortexOccipital cortexMedial cortexVisual deprivationEmission tomographyTransectionCommissure
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