2023
Pathogenesis and Prediction of Preterm Delivery
Kendle A, Buhimschi C, Lockwood C. Pathogenesis and Prediction of Preterm Delivery. 2023, 344-360. DOI: 10.1002/9781119636540.ch40.Peer-Reviewed Original ResearchPreterm birthPR expressionPlacental corticotropin-releasing hormonePrediction of PTBAdrenal (HPA) axis pathwayCorticotropin-releasing hormoneToll-like receptorsProtease-activated receptorsPreterm deliveryProgesterone receptor inhibitorsDecidual hemorrhageInflammatory cascadeProinflammatory cytokinesInflammatory chemokinesProgesterone activityInterleukin-1βSonographic markersReceptor inhibitorsCortisol releaseInflammation pathwaysMatrix metalloproteinasesAxis pathwayPremature activationBiochemical remodelingProgressive withdrawal
2002
Acute citalopram administration produces correlated increases in plasma and salivary cortisol
Bhagwagar Z, Hafizi S, Cowen P. Acute citalopram administration produces correlated increases in plasma and salivary cortisol. Psychopharmacology 2002, 163: 118-120. PMID: 12185409, DOI: 10.1007/s00213-002-1149-4.Peer-Reviewed Original ResearchConceptsSalivary cortisol levelsCortisol levelsSelective serotonin re-uptake inhibitorsSalivary cortisolSerotonin re-uptake inhibitorsAcute citalopram administrationRe-uptake inhibitorsCross-over designSalivary cortisol concentrationsCitalopram administrationIntravenous administrationCortisol releaseHealthy volunteersCortisol concentrationsSaliva cortisolCitalopramCortisolParallel increaseAdministrationCorrelated increaseAcceptable means
2001
Relationship Among Plasma Cortisol, Catecholamines, Neuropeptide Y, and Human Performance During Exposure to Uncontrollable Stress
Morgan C, Wang S, Rasmusson A, Hazlett G, Anderson G, Charney D. Relationship Among Plasma Cortisol, Catecholamines, Neuropeptide Y, and Human Performance During Exposure to Uncontrollable Stress. Biopsychosocial Science And Medicine 2001, 63: 412-422. PMID: 11382268, DOI: 10.1097/00006842-200105000-00010.Peer-Reviewed Original ResearchConceptsPosttraumatic stress disorderStress disorderPsychobiological responsesUncontrollable stressThreat response systemStress-related illnessesIndividual differencesTrauma exposurePsychological dissociationAversive experiencesNeurobiological factorsHuman performanceBehavioral indicesSurvival SchoolNeurobiological perturbationsNeurobiological responsesPsychological symptomsCatecholamine functioningMilitary performanceDisordersNeuropeptide YCortisol releaseIndividualsAdrenocortical axisNeuropeptide Y release
2000
Effects of Caffeine on Muscle Glycogen Utilization and the Neuroendocrine Axis during Exercise1
Laurent D, Schneider K, Prusaczyk W, Franklin C, Vogel S, Krssak M, Petersen K, Goforth H, Shulman G. Effects of Caffeine on Muscle Glycogen Utilization and the Neuroendocrine Axis during Exercise1. The Journal Of Clinical Endocrinology & Metabolism 2000, 85: 2170-2175. PMID: 10852448, DOI: 10.1210/jcem.85.6.6655.Peer-Reviewed Original ResearchConceptsMuscle glycogen contentMuscle glycogen utilizationGlycogen contentCaffeine ingestionNeuroendocrine axisGlycogen utilizationGlycogen-sparing effectFree fatty acid concentrationsBeta-endorphin levelsCaffeine-treated groupExercise-induced glycogen depletionMaximal oxygen consumptionEffects of caffeineHigher muscle glycogen contentPlacebo groupExercise enduranceFatty acid concentrationsPlasma concentrationsNeuroendocrine hormonesCortisol releaseProlonged exerciseGlycogen depletionPlasma lactateNormal valuesThigh muscles
1999
Naloxone Challenge in Smokers: Preliminary Evidence of an Opioid Component in Nicotine Dependence
Krishnan-Sarin S, Rosen M, O'Malley S. Naloxone Challenge in Smokers: Preliminary Evidence of an Opioid Component in Nicotine Dependence. JAMA Psychiatry 1999, 56: 663-668. PMID: 10401515, DOI: 10.1001/archpsyc.56.7.663.Peer-Reviewed Original ResearchConceptsEndogenous opioid systemOpioid systemNicotine dependenceOpiate-like withdrawalNicotine-dependent individualsDose-dependent increaseNicotine-dependent subjectsPreliminary evidenceOpioid componentAdrenal axisNaloxone challengeWithdrawal signsCortisol releaseNeuroendocrine parametersNicotine cravingCigarette smokeLong-term exposureCortisol levelsBaseline levelsLow dosesSubjective signsNaloxoneSmokersChallenge proceduresSymptoms
1991
Opiate blockade enhances hypoglycemic counterregulation in normal and insulin-dependent diabetic subjects
Caprio S, Gerety G, Tamborlane WV, Jones T, Diamond M, Jacob R, Sherwin RS. Opiate blockade enhances hypoglycemic counterregulation in normal and insulin-dependent diabetic subjects. American Journal Of Physiology 1991, 260: e852-e858. PMID: 2058661, DOI: 10.1152/ajpendo.1991.260.6.e852.Peer-Reviewed Original ResearchConceptsInsulin-induced hypoglycemiaGlucose counterregulationIDDM patientsGlucose productionOpiate blockadeHormonal responsesInsulin-dependent diabetic subjectsCounterregulatory hormone releaseDefective glucose counterregulationInsulin clamp studiesAbsence of hypoglycemiaInsulin-dependent patientsVariable glucose infusionDefective counterregulationHypoglycemic counterregulationNaloxone studyGlucagon responseDiabetic subjectsEndogenous opiatesGlucoregulatory hormonesGlucoregulatory responsesHormone releaseGlucose infusionHealthy subjectsCortisol release
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