2016
Repeated measures analysis of associations between urinary bisphenol-A concentrations and biomarkers of inflammation and oxidative stress in pregnancy
Ferguson K, Cantonwine D, McElrath T, Mukherjee B, Meeker J. Repeated measures analysis of associations between urinary bisphenol-A concentrations and biomarkers of inflammation and oxidative stress in pregnancy. Reproductive Toxicology 2016, 66: 93-98. PMID: 27751756, PMCID: PMC5125888, DOI: 10.1016/j.reprotox.2016.10.002.Peer-Reviewed Original ResearchConceptsInterquartile range increaseBisphenol AOxidative stressRange increaseUrinary bisphenol A concentrationsC-reactive proteinBiomarkers of inflammationIL-6 concentrationsAdverse birth outcomesPrimary mechanism of actionAssociated with significant increasesRepeated measures analysisBisphenol A exposureBiomarkers of oxidative stressBisphenol A concentrationsMechanism of actionPregnant womenFetal developmentImpact pregnancyInflammation markersBirth outcomesOxidative stress biomarkersIL-6BPA exposurePregnancy
2015
Urinary Bisphenol A Levels during Pregnancy and Risk of Preterm Birth
Cantonwine D, Ferguson K, Mukherjee B, McElrath T, Meeker J. Urinary Bisphenol A Levels during Pregnancy and Risk of Preterm Birth. Environmental Health Perspectives 2015, 123: 895-901. PMID: 25815860, PMCID: PMC4559950, DOI: 10.1289/ehp.1408126.Peer-Reviewed Original ResearchConceptsRisk of preterm birthPreterm birthBisphenol A exposurePreterm premature rupture of membranesClinical classificationClassification of preterm birthSubcategories of preterm birthPremature rupture of membranesBisphenol A LevelsCases of preterm birthAssociated with preterm birthSpontaneous preterm laborPreterm premature ruptureRupture of membranesSpontaneous preterm birthIntrauterine growth restrictionNested Case-Control StudyProspective birth cohort studyUrinary bisphenol ACase-control studyAssociated with increased oddsA levelsTertiary teaching hospitalBirth cohort studyDelivered preterm
2013
Bisphenol A delays the perinatal chloride shift in cortical neurons by epigenetic effects on the Kcc2 promoter
Yeo M, Berglund K, Hanna M, Guo J, Kittur J, Torres M, Abramowitz J, Busciglio J, Gao Y, Birnbaumer L, Liedtke W. Bisphenol A delays the perinatal chloride shift in cortical neurons by epigenetic effects on the Kcc2 promoter. Proceedings Of The National Academy Of Sciences Of The United States Of America 2013, 110: 4315-4320. PMID: 23440186, PMCID: PMC3600491, DOI: 10.1073/pnas.1300959110.Peer-Reviewed Original ResearchMeSH KeywordsAir Pollutants, OccupationalAnimalsBenzhydryl CompoundsCells, CulturedCentral Nervous System DiseasesCerebral CortexChloridesDNA-Binding ProteinsEpigenesis, GeneticFemaleHistone Deacetylase 1HumansK Cl- CotransportersMaleMiceNerve Tissue ProteinsNeuronsPhenolsRatsResponse ElementsSex CharacteristicsSymportersConceptsEffects of bisphenol AKcc2 promoterBisphenol A exposureKCC2 expressionCortical neuronsHistone deacetylase 1Binding of methyl-CpG-binding protein 2Potassium-chloride cotransporter 2KCC2 gene expressionMethyl-CpG-binding protein 2Knockdown of histone deacetylase 1Sexually dimorphic mannerSexually dimorphic effectsHuman neurodevelopmental disordersBisphenol-A effectsHuman cortical neuronsToxic mechanisms of bisphenol ANeuronal chlorideCotransporter 2Cortical slicesBinding protein 2KCC2Mouse damsDimorphic mannerInterneuron migration
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