2004
Insulin resistance in thermally-injured rats is associated with post-receptor alterations in skeletal muscle, liver and adipose tissue.
Carter E, Burks D, Fischman A, White M, Tompkins R. Insulin resistance in thermally-injured rats is associated with post-receptor alterations in skeletal muscle, liver and adipose tissue. International Journal Of Molecular Medicine 2004, 14: 653-8. PMID: 15375597, DOI: 10.3892/ijmm.14.4.653.Peer-Reviewed Original ResearchConceptsUrinary C-peptide excretionC-peptide excretionPost-receptor alterationsInsulin resistanceInsulin receptor bindingSkeletal muscleInsulin infusionBurn injuryAdipose tissueFull-thickness scald injuryGlucose productionSham-treated control animalsReceptor bindingHepatic glucose productionIRS-1 expressionWestern blot methodBinding of insulinAbsence of changesScald injuryBolus injectionRat modelPossible molecular mechanismsControl animalsInjuryThermal injury
1985
Normal hepatic insulin receptor autophosphorylation in nonketotic diabetes mellitus
Amatruda J, Roncone A. Normal hepatic insulin receptor autophosphorylation in nonketotic diabetes mellitus. Biochemical And Biophysical Research Communications 1985, 129: 163-170. PMID: 3890853, DOI: 10.1016/0006-291x(85)91417-2.Peer-Reviewed Original ResearchConceptsHepatic insulin resistanceNonketotic diabetes mellitusInability of insulinDiabetes mellitusInsulin resistanceInsulin receptor autophosphorylationInsulin actionPutative mediatorsStreptozotocin-induced diabetes mellitusReceptor autophosphorylationDose-response relationshipInsulin receptor bindingAbility of insulinMembrane insulin receptorsDiabetic animalsInsulin treatmentInsulin dose-response relationshipInsulin bindingMellitusPlasma membrane insulin receptorsInsulinReceptor bindingInsulin receptor
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