2019
Application of the Milan System for Reporting Submandibular Gland Cytopathology: An international, multi‐institutional study
Maleki Z, Baloch Z, Lu R, Shafique K, Song SJ, Viswanathan K, Rao RA, Lefler H, Fatima A, Wiles A, Jo VY, Wang H, Fadda G, Powers CN, Ali SZ, Pantanowitz L, Siddiqui MT, Nayar R, Klijanienko J, Barkan GA, Krane JF, Rossi ED, Callegari F, Kholová I, Bongiovanni M, Faquin WC, Pusztaszeri MP. Application of the Milan System for Reporting Submandibular Gland Cytopathology: An international, multi‐institutional study. Cancer Cytopathology 2019, 127: 306-315. PMID: 31050186, PMCID: PMC7404554, DOI: 10.1002/cncy.22135.Peer-Reviewed Original ResearchAdolescentAdultAgedAged, 80 and overAlgorithmsBiopsy, Fine-NeedleChildChild, PreschoolCytodiagnosisFemaleFollow-Up StudiesHealth FacilitiesHumansInfantInternational AgenciesMaleMedical RecordsMiddle AgedPrecancerous ConditionsRetrospective StudiesRisk AssessmentSalivary Gland NeoplasmsSubmandibular GlandYoung Adult
2017
“Suspicious” salivary gland FNA: Risk of malignancy and interinstitutional variability
Maleki Z, Miller JA, Arab SE, Fadda G, Bo P, Wise O, Rossi ED, Jhala N, Ashish C, Ali SZ, Wang H. “Suspicious” salivary gland FNA: Risk of malignancy and interinstitutional variability. Cancer Cytopathology 2017, 126: 94-100. PMID: 29053216, DOI: 10.1002/cncy.21939.Peer-Reviewed Original ResearchConceptsRisk of malignancySalivary gland FNAsFNA specimensExact testSuspicious casesFine needle aspiration cytologyEpithelial metaplastic changesSignificant interinstitutional variabilityTertiary medical centerFisher's exact testSalivary gland neoplasmsSalivary gland lesionsSFM categoryMetaplastic changesMalignancy categoryConclusive diagnosisMalignant tumorsAspiration cytologyMedical CenterFNA casesIndeterminate resultsSFM groupGland neoplasmsSpecific neoplasmsGland lesions
2013
Altered vascular activation due to deficiency of the NADPH oxidase component p22phox
Wang H, Albadawi H, Siddiquee Z, Stone J, Panchenko M, Watkins M, Stone J. Altered vascular activation due to deficiency of the NADPH oxidase component p22phox. Cardiovascular Pathology 2013, 23: 35-42. PMID: 24035466, DOI: 10.1016/j.carpath.2013.08.003.Peer-Reviewed Original ResearchMeSH KeywordsAnimalsCarotid Artery InjuriesCarotid Artery, CommonCase-Control StudiesCasein Kinase IalphaCells, CulturedCoronary VesselsCytochrome b GroupElastic TissueFemaleGranulomatous Disease, ChronicHeterogeneous-Nuclear Ribonucleoprotein Group CHumansHyperplasiaInfantMaleMatrix Metalloproteinase 12MiceMice, KnockoutMuscle, Smooth, VascularNADPH OxidasesNeointimaReactive Oxygen SpeciesRNA InterferenceTissue Inhibitor of Metalloproteinase-1TransfectionConceptsWild-type littermatesElastic fiber lossVascular activationDeficient miceIntimal hyperplasiaNADPH oxidase complexCoronary artery smooth muscle cellsNicotinamide adenine dinucleotide phosphate (NADPH) oxidaseArtery smooth muscle cellsHuman coronary artery smooth muscle cellsAdenine Dinucleotide Phosphate OxidaseBasal blood pressureCarotid artery ligationExpression ratioSmooth muscle cellsLung biopsyBlood pressureFiber lossArtery ligationCarotid ligationCarotid arteryTissue inhibitorHuman patientsMuscle cellsMice