AMP‐Activated Protein Kinase Attenuates High Salt‐Induced Activation of Epithelial Sodium Channels (ENaC) in Human Umbilical Vein Endothelial Cells
Zheng W, Li X, Liu H, Wang Z, Hu Q, Li Y, Song B, Lou J, Wang Q, Ma H, Zhang Z. AMP‐Activated Protein Kinase Attenuates High Salt‐Induced Activation of Epithelial Sodium Channels (ENaC) in Human Umbilical Vein Endothelial Cells. Oxidative Medicine And Cellular Longevity 2016, 2016: 1531392. PMID: 27635187, PMCID: PMC5011216, DOI: 10.1155/2016/1531392.Peer-Reviewed Original ResearchConceptsAMP-activated kinaseAMP-activated kinase activityHuman umbilical vein endothelial cellsEpithelial sodium channelAMP-activated protein kinaseUmbilical vein endothelial cellsEndothelial cellsVein endothelial cellsActivity of epithelial sodium channelsCell-attached patch-clamp techniqueSodium channelsNO productionHigh-sodiumENOS phosphorylationProtein kinaseENaC open probabilityEffects of high sodiumPatch-clamp techniqueSodium treatmentPhosphorylationHigh saltNitric oxide synthaseExpression levelsENaC blockersENaC expression
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